Interpreting HSAT Like a Pro: AHI, ODI, T90, REM and Positional Effects

Interpreting HSAT Like a Pro: AHI, ODI, T90, REM and Positional Effects

HSAT is a convenient first test for suspected OSA, but it estimates sleep without EEG and can miss severity. Look beyond AHI to avoid under-treating.

1) AHI vs ODI—don’t stop at one number

  • AHI = events/hour, but on HSAT it’s against recording time, so severity can be underestimated (e.g., insomnia, fragmented sleep).

  • ODI = oxygen drops/hour (≥3–4%). A high ODI with borderline AHI signals meaningful hypoxic burden and may still merit treatment or PSG.
    Rule of thumb: If ODI > AHI pattern, think “oxygen-burden phenotype”—so don’t dismiss it.

2) REM and position—simple patterns that change management

  • REM-predominant clues: clusters later in the night or deeper desaturations without a position change. These events run longer and drop O₂ more.

  • Positional OSA: events mainly supine. Positional therapy may help, but CPAP remains first-line for moderate–severe disease.

3) When a “mild” AHI isn’t mild

Borderline AHI (5–15) still matters if the patient has:

  • Symptoms: daytime sleepiness, fatigue, snoring with witnessed apnoeas

  • Comorbidities: HTN, AF, metabolic disease

  • Safety-sensitive roles: drivers, heavy machinery, pilots. Consider treatment or PSG rather than “watchful waiting.”

4) Red flags → escalate to PSG/specialist

  • Inadequate/inconclusive HSAT or strong clinical suspicion despite a negative study

  • High T90 (e.g., >20% of recording) or frequent desat clusters with modest AHI

  • Mismatch between position data and symptoms

  • Complexity/comorbidities: heart failure, COPD, neuromuscular disease, opioid use, stroke, insomnia features, suspected hypoventilation

5) Quick next-step guide

  • High AHI + high ODI: classic OSA → start CPAP (plus long-term risk factor management).

  • High ODI, borderline AHI: oxygen-burden phenotype → treat or confirm with PSG.

  • Supine clustering: positional therapy ± CPAP based on severity.

  • Late-night clusters: possible REM-predominant → consider PSG if unclear.

  • Borderline AHI + symptoms/comorbidities/safety risk: PSG.

  • Inconclusive/poor-quality study or complex comorbidity: PSG/specialist.

How we can help: We provide streamlined HSAT with physician-interpreted reports and practical next-step guidance. When indicated, we coordinate escalation to in-lab PSG and support CPAP setup and follow-up so patients don’t fall through the cracks.

Key references: 

Kapur VK, Auckley DH, Chowdhuri S, et al. Clinical Practice Guideline for Diagnostic Testing for Adult Obstructive Sleep Apnea. J Clin Sleep Med. 2017;13(3):479–504. AASM Guidelines

Royal Australian College of General Practitioners (RACGP). Obstructive sleep apnoea in adults: Clinical update. AJGP. June 2024. RACGP 2024 Guidance

Azarbarzin A, et al. The hypoxic burden of sleep apnoea predicts cardiovascular disease-related mortality. Eur Heart J. 2019;40(14):1149–1157. DOI: 10.1093/eurheartj/ehy624

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